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GCG activation suppresses endogenous hepatic lipid production by inhibiting lipogenic enzymatic pathways while simultaneously enhancing mitochondrial oxidative lipid catabolism, thereby promoting lipid catabolism and reducing hepatic triglyceride content [18]

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BGL in general substantially dropped from pre-treatment to pre-S1 (F 1,4 = 8.79, p = 0.041), change-score analysis further analyses are baseline-adjusted models or raw values

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