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An earlier short-term randomized trial did not find meaningful changes in several oxidative-stress markers after oral glutathione

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Some studies have revealed that the antioxidative mechanism of VD may involve reducing the expression and activity of Pin1 protein by activating VDR, preventing the Ser-36 phosphorylation and mitochondrial translocation of p66Shc induced by PKC-, inhibiting mitochondrial oxidative stress and inflammation, and improving endothelial function [136]