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Studies have shown that CaMK upregulates and activates PGC-1 by promoting the phosphorylation of p38 mitogen-activated protein kinases (MAPK) and activating extracellular regulated protein kinase (ERK)/cAMP-response element-binding protein (CREB) pathways [43, 45, 46]
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In preclinical research, BPC-157 is typically reconstituted in bacteriostatic water and administered via intraperitoneal injection in rodent models
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