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Lp(a) assembly begins with Apo(a) docking to low-density lipoprotein (LDL), followed by formation of a covalent disulfide bond between Kringle IV-type 9 of Apo(a) and ApoB of LDL, as shown in Figs

As succinate accumulates via the inhibition of the 2-OGDD enzyme, epigenetic modification acts in the process of cell transformation into a hypermethylated phenotype 100

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Patients with asymptomatic gallstones (silent stones discovered incidentally on imaging) present a nuanced scenario

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