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No large human trial has yet clarified its in vivo mechanism, a translational gap flagged in current commentary [6]

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Importantly, these expanded HBs retained bipotency, co-expressing both hepatic and early cholangiocyte markers [1]

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The honest position is this: there is no head-to-head human bioavailability trial telling you which route is superior for this specific molecule, so the best route is not a settled question