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30 min pretreatment, followed by 6-24 h of AO exposure) inhibits AO-induced activation of the NF-B signaling pathway in BV-2 cells, upregulates and maintains the expression of Nurr1 mRNA and protein, and thereby exerts anti-inflammatory effects by suppressing the binding of NF-B p65 to the iNOS promoter induced by AO in BV-2 cells [5]