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Although siRNA studies show that GSTO1 is important for HCT116 cancer cell viability, knockdown of GSTO1 did not appear to affect C1-27 cytotoxicity, suggesting that other targets of C1-27 also may contribute to its cytotoxicity and have a compensatory effect (Supplementary Fig

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IL-17A blockade faces redundancy at multiple levels: IL-17F and IL-17A/F heterodimers signal through the same receptor complex (73), and TNF-/IL-1-driven NF-B activation maintains parallel neutrophil recruitment independently of IL-17A ( Translating pathophysiological mechanisms into clinical benefits requires addressing the limitations of current COPD treatments