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Although biochemically separated from the GSH system, the Trx system shares NADPH as a common reducing cofactor and serves as an alternative pathway for reducing disulfide bonds, making this system especially relevant if the GSH system has been compromised or rendered dysfunctional (Silva-Adaya et al., 2014)

Endothelial dysfunction is distinguished by reduced NO bioavailability and reduced vasodilation connected to a proinflammatory and prothrombotic condition

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