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Taken together, these results suggest that Gln-derived aKG is an important cofactor required for histone modification and that decreased Gln-derived aKG production suppresses histone demethylation, specifically H3K9me3 demethylation, by inhibiting KDM4C activity, leading to significant downregulation of cFLIP expression

10.3389/fphys.2017.00600 Front Physiol