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The peptide affects genes involved in tissue remodeling, antioxidant defense, DNA repair, inflammation control, and cellular stress responses

Gerhart D, LeVasseur R, Broderius M, Drewes LR

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In animal models it repaired cognitive deficits at extraordinarily low doses but no human trials of any kind have been published, and its long-term safety is entirely unknown

The administration of cysteine-rich glutathione precursors, especially N-acetyl cysteine, has been shown to increase intracellular glutathione levels

In the context of diabetes models, it may contribute to heart attack, impaired wound healing, stroke, and kidney damage