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Reticulon-1A mediates diabetic kidney disease progression through endoplasmic reticulum-mitochondrial contacts in tubular epithelial cells

Mancini GBJ, OMeara E, Zieroth S, Bernier M, Cheng AYY, Cherney DZI, et al

Beaudry, J

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This hypothesized systemic dissemination of inflammation constitutes the mechanistically plausible pathophysiological bridge between testicular dysfunction and penile ED, a mechanism that has long been underrecognized in traditional research focusing solely on local testicular changes (204)