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While standard therapies, such as glucocorticoids, beta-2 adrenergic receptor (2AR) agonists, and biologics (e.g., anti-IgE, anti-IL-5, anti-IL5R, anti-IL4R) are effective in T2 high asthma, their efficacy is often diminished or non-existent in T2 low or metabolically driven asthma phenotypes

The NF-B pathway functions as a central pro-survival and pro-inflammatory transcription factor that promotes tumor progression by suppressing tumor suppressors such as phosphatase and tensin homolog (PTEN), thereby indirectly enhancing PI3K/AKT signaling and supporting cancer cell viability

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