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found that glutamine and its dipeptides restored oxidative metabolism in neonatal hepatocytes impaired by septic mediators (H 2 O 2 , NO), likely through boosting glutathione synthesis rather than the Krebs cycle
In parallel, excessive QA-driven NMDAR activation impairs sarco/endoplasmic reticulum Ca-ATPase function, disrupting calcium homeostasis and intracellular signalling [148]
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This study delves into TNBCs tumour microenvironment (TME) remodeling, therapeutic resistance, and immunoengineering strategies using nanotechnology
However, further studies are needed to confirm their efficacy and clarify whether their therapeutic effects are truly mediated through antioxidant mechanisms
Mei for her help in producing this manuscript
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