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The integration of kinase (PKA, CDK5, GSK3) and small G-protein (IQGAP1) signalling pathways through CaMKK2, all of which have demonstrated links with human bipolar disorder, points to the possibility that bipolar disorder is a signalopathy that stems from defects in this signal transduction network, and potentially explains the clinical heterogeneity and polygenic nature of the condition
Oxidative brain damage in Mecp2-mutant murine models of Rett syndrome
Each measurement was carried out in triplicate ( n = 3), and results were expressed as mean standard deviation (SD)
The observational findings corroborate earlier SELECT trial results but do not establish causation, making further prospective research essential
If you experience any suspected side effects, report them via the MHRA Yellow Card Scheme (yellowcard.mhra.gov.uk)
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