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The exact mechanism of resistance offered and the systemic implications of glutathione supplementation need to be sought out but most of the reactions are supposed to be a manifestation of its antioxidative property

DNA methylation-driven EMT is a common mechanism of resistance to various therapeutic agents in cancer

To evaluate whether mutants deficient in different glutathione-dependent enzymes are impacted by TMHT, we subjected cytosolic glutaredoxin mutants to TMHT (Figure 4C)

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