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Furthermore, recent studies have particularly emphasized the close association between IgA nephropathy and gut microbiome dysregulation

When IgG auto-Abs bind to a cell-surface target, they can activate complement or immune cell-mediated inflammatory responses directed against non-pathogenic tissue [20, 131]

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In later stages, chronic A exposure or inflammation may induce tolerance, reducing cytokine release and promoting repair, though this response can be maladaptive or insufficient