It is better understood as one possible biochemical contributor that may amplify oxidative stress, mitochondrial dysfunction and medication sensitivity
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In stark contrast, significant chromosome damage was evident upon treatment with the DNA crosslinking agent MMC, thus suggesting that when DNA repair is functional, FA might be causing cell death by damaging other cellular components rather than DNA
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Deepika Shukla (Scientist, NINS laboratory) and Ms
Gholinejad M, Anarkooli IJ, Taromchi A, Abdanipour A (2018) Adenosine decreases oxidative stress and protects H2O2-treated neural stem cells against apoptosis through decreasing Mst1 expression