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The non-enzymatic mitochondrial protein hyperacetylation induced by the increased availability of mitochondrial acetyl-CoA avoids the risk associated with mitochondria overfeeding, restraining the activity of the respiratory complex I and modulating CACT activity

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In experiments using cultured cells, it was reported that PQQ Na 2 prevents oxidative stress-induced neuronal death.Citation 76,77 ) It has been shown that PQQ prevented 6-hydroxydopamine (6-OHDA)-induced cell death of the dopaminergic neuroblastoma cell line SH-SY5Y and primary rat neurons and that its preventive effect was stronger than that of vitamin C and E.Citation 76 ) 6-OHDA is a well-known neurotoxin that compromises mitochondria complex I, resulting in the production of ROS, such as , hydroxyl radicals, and H 2 O 2

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