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Glucose-6-phosphate is oxidized to 6-phosphogluconolactone by glucose-6-phosphate dehydrogenase (G6PD) with the transfer of two electrons to NADP+ to form NADPH

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It has also been confirmed that AD pathologic markers, oligomeric A and A plaque load, could be reduced by LG and microglial activation is decreased and memory behaviors are improved in APP swe /PS1_E9 (APP/PS1) mouse model after using LG (74)

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In vitro experiments confirmed that A 25-35 treatment significantly inhibited the activation of the NRF2/SLC7A11/GPX4 signaling pathway in HT22 cells, upregulated the expression of Fe 2+ , TFR1, ACSL4, MDA, and GSSG, and decreased the expression of FTH1, SOD, and GSH, as well as the GSH/GSSG ratio, resulting in dysregulated neuronal iron metabolism, impaired amino acid antioxidant systems, mitochondrial dysfunction, and ferroptosis, which ultimately promoted neuronal injury