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Mitochondrial dysfunction in Parkinson's disease: molecular mechanisms and pathophysiological consequences

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Researchers often study GHK-CU for its potential relevance to: Collagen-related signaling Tissue remodeling Skin regeneration support Fibroblast activity Extracellular matrix regulation Anti-inflammatory and antioxidant pathways Several published studies have associated GHK and GHK-CU with tissue repair, skin remodeling, and regenerative biological pathways

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Neurological manifestations are particularly concerning and may include peripheral neuropathy with paresthesias (tingling and numbness in hands and feet), ataxia, impaired proprioception and vibratory sense, cognitive impairment, memory loss, and in severe cases, subacute combined degeneration of the spinal cord

doi:10.1186/1550-2783-4-22