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These salt bridges stabilize the ligand-receptor complex and improve binding affinity and activation, providing a robust template for the development of next-generation GLP-1R agonists
doi: 10.1002/path.4304 112 WitzelIOliveira-FerrerLPantelKMullerVWikmanH
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2020;5:e135418
195 Moreover, in CTSL-deficient mice, both trypsin activity and pancreatic apoptosis were increased, yet disease severity was paradoxically reduced
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