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Commonly, long-acting ones show a more powerful glucose lowering effect as it lowers not only postprandial glucose levels but also fasting glucose levels [23]

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The question: does sustained elevation of intracellular NAD+ via NNMT inhibition produce the same long-term benefits as NAD+ precursor supplementation (like nicotinamide riboside or NMN), or are there trade-offs we haven't identified yet

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