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First, mechanistic studies should integrate single-cell spatial transcriptomics and mitochondrial metabolomics to map ferroptosis pathways in OPCs, mature oligodendrocytes, and microglia within periventricular white matter of infants with PVL

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Dose-dependently reduced hepatic CD36, PPAR, and CPT1A protein expression, and increased hepatic p-IGF-1R, p-IRS1, p-PI3K, and p-Akt protein expression in db/db mice

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