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Several enzymes involved in the glutathione pathway, such as -glutamylcysteine synthetase (GCLC), glutathione synthetase (GSS), glutathione peroxidase 4 (GPX4), and glutathione reductase (GSR), are regulated, at least in part, by Nrf2 through its activation of the Nrf2-antioxidant response element

Our current understanding is that both, circulating IGF-I levels and locally produced IGF-I, contribute to the maintenance of microcirculation functional and structural integrity, increasing NO bioavailability, decreasing reactive oxygen species (ROS) production, and exerting antiinflammatory, antiapoptotic, and pro-angiogenic effects

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