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142 RosboroughK.PatelN.KaliaL
Taken together, the above data demonstrate that GLP-1(2836) acts through a molecular pathway involving the catalytic generation of cAMP from sAC in cardiac vascular cells to preserve membrane integrity and prevent apoptosis following oxidative injury
& Calabr, P
P=NS), confirming anti-fibrotic rather than anti-inflammatory mechanism at this dose
The FDA label notes reports of acute kidney injury in patients treated with GLP-1 receptor agonists, predominantly in patients who experienced volume depletion from GI side effects [6]
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