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They work through several mechanisms that are relevant even when your pancreas no longer makes insulin: they slow gastric emptying (meaning food hits your bloodstream more gradually), suppress glucagon release (the hormone that raises blood sugar), and reduce appetite
The distribution might also cross certain zones and may manifest as bridging necrosis showing confluence of the lesions (e.g., central to central veins, portal tract to portal tract, or portal tract to central zones)
You can adjust your dosage or timing based on what you observe
Imagine the healing process as a three-layer phase: Layer 1: Structural Activation (TB-500) Increases cell mobility Guides fibroblasts and endothelial cells Drives angiogenesis Organizes the extracellular matrix Layer 2: Biochemical Stabilization (BPC-157) Maintains vascular tone through nitric-oxide regulation Protects endothelial integrity Supports nerve healing Modulates inflammation to prevent delays Layer 3: Overlap Zone Both enhance angiogenesis Support fibroblast activity Both contribute to microcirculation restoration When the layers stack, you get a model in which TB-500 drives the action , while BPC-157 controls the environment
