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[5] Peeling, P., Binnie, M

How it works The best-documented mechanism runs through VEGFR2, a receptor that triggers new blood vessel formation, paired with the Akt-eNOS pathway, which generates nitric oxide to support vascular repair and restore blood flow to damaged tissue (Rodgers et al., 2025)

This duality is particularly evident in LKB1-deficient cancers, where AMPK is inactive