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Besides an increased homocysteine export from tissues into plasma that may be regulated by a reduced-homocysteine carrier (Blom, 2000), hyperhomocysteinemia can result from reduced renal function, which may have a central role in plasma homocysteine clearance, and from chronic hepatic insufficiency
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Activation of specific heterotrimeric G proteins involves different regions of the receptors intracellular loops, primarily the third intracellular loop (8, 50, 68)
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Therefore, the addition of glucose-lowering agents is recommended by the American and European Diabetes Associations to control and/or minimize the risk of cardiovascular disease and microvascular complications [103]
