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The 2008 review touched on hair-follicle work as part of the wider tissue-remodelling discussion (PMID 18644225)
Endocr J (2016) 63(6):58996
Related reading: toxic overload, elevated SAH and impaired methylation
777 The mechanism may be related to the activation of transcription factor SATB1, upregulation of the SLC7A11/HO-1 signaling pathway, and improvement of neuronal ferroptosis
The body does not exclusively target fat stores during periods of severe energy restriction
Beyond that window, oxidative degradation accelerates and potency drops
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