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Indeed, ATP availability in these mutant animals was reduced by roughly threefold the levels observed in wild-type animals after fasting (Supplementary Fig
Protein oxidation in aging and the removal of oxidized proteins
& Han, W
KEAP1/NRF2 mutant cells are protected against cysteine-starvation induced ferroptosis due to elevated GCLC expression and limited glutamate accumulation, and dual targeting of cystine import and GCLC activity can overcome the resistance of NRF2 active cells to ferroptosis (Kang et al., Cell Metab 2021)
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