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Myotendinous Junction Repair (Japjec et al., 2021) In myotendinous-junction injuries, BPC-157 led to full defect closure and functional restoration within six weeks, normalizing nitric-oxide pathways and revascularization

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These metabolic issues can disrupt the body's energy utilization, lead to fat accumulation, increase metabolic burden, and are detrimental to post-infarction recovery, even raising the risk of complications during the recovery period

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