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However, the literature does not yet provide a fully resolved primary molecular target, a unified causal mechanism, or sufficient controlled human evidence to support conclusions about therapeutic use
2020 Dec;6(4):161-172
Bonomini M, Zammit V, Pusey CD, De Vecchi A, Arduini A
J., & Bray, R
The resulting inflammatory mediators impair bloodbrain barrier function ( Figure 4 ), facilitating the entry of peripheral cytokines and gut-derived metabolites, including trimethylamine N-oxide, into the brain
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