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GLP-1 activation can reduce the pathological process of amyloid- plaque aggregation/deposition and tau hyperphosphorylation enhanced by neuroinflammation, thus preventing age-related neurodegenerative changes, such as decline of learning and memory (Li et al., 2012)
Il a la capacit damliorer les fonctions crbralescar il peuttraverser la membrane protectrice du cerveau (barrire hmato-encphalique), signifiant quelle peut augmenter le mtabolisme nergtique des cellules du cerveau
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10.1161/circulationaha.107.735795 55 JastreboffA
As the figure shows, both GLP-1RAs and SGLT-2is could reduce the MACE, with the SGLT-2is treatment seem to be nominally associated with lower risk of heart failure and total mortality, whereas with GLP-1RAs seem to be associated with lower risks of stroke and peripheral artery disease
Immune cells Some research suggests that semaglutide may help reduce oxidative stress (which can cause cell damage)and inflammation, both of which may contribute to obesity
