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But excessive or prolonged inflammation becomes destructive

An early publication in the Annals of the New York Academy of Sciences documenting the original discovery of telomere shortening as a primary mechanism of cellular senescence and its relevance to human aging biology provides the foundational framework for understanding why telomerase-activating compounds attract longevity research attention

Presence of a mutation in PSEN1 or PSEN2 gene is associated with an impaired brain endothelial cell phenotype in vitro

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Pericyte ablation thoroughly leads to acute BBB breakdown and severe loss of CBF [101]

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