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Chronobiol Int
Interestingly, oxidative and nitrosative stress can also inactivate parkin by inducing post-translational protein modifications and misfolding with the subsequent accumulation of PARIS, and may also accelerate -synuclein aggregation, which together may account for mitochondrial dysfunction (West and Maidment, 2004
F.VuralA.KkoluK.et al (2019)
Its currently under investigation and lacks approval for therapeutic use in humans
People with documented MTHFR variants or labs suggesting impaired methylation
But all too often, drugs that show promise in experimental animals simply dont perform very well once theyre tested in humans
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