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Strategies that deplete GSH, such as using inhibitors like BSO which is inhibitor of -glutamylcysteine synthetase (-GCS) , the rate-limiting enzyme in glutathione (GSH) synthesis or by combining prodrug activation with GSH depletion in nanosystems, have shown promise in overcoming MDR and improving chemotherapy effectiveness [72]
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These results guide the concurrent hormonal rebalancing required for full recovery
Multi-omics analysis identifies ATF4 as a key regulator of the mitochondrial stress response in mammals
Meanwhile, by downregulating AIF-1/TRPC6/calcineurin A/NFAT2 axis, miR-30a regulates pyroptosis and inflammatory responses of TECs during DKD (185)
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