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U., Cinatl, J., Dauletbaev, N., Buxbaum, S., Treusch, G

However, studies to investigate the therapeutic potential of GHR antagonism in these diseases have been limited, most likely due to difficulty in accessing therapeutic tools to study the pharmacology of the receptor in vivo

Since then, it has been the subject of extensive research

The Pyo 2007 study (PMID 17703734) reported that AHK-Cu raised VEGF production in the treated cells

They are different molecules with different mechanisms, which is exactly why researchers often look at them side by side

This guesswork can be dangerous, as the line between a helpful dose and a harmful one is completely undefined