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Anthracyclines Anthracycline drug family discovery is dated in the 1950s with daunorubicin isolation from Streptomyces peucetius ( Anthracycline anti-tumor effect depends on several mechanisms such as apoptosis induction via inhibition of topoisomerase II (TOP2), intercalation into the deoxyribonucleic acid (DNA) leading to an inhibition of macromolecules synthesis, or production of reactive oxygen species (ROS) causing DNA damage or lipid peroxidation ( Anti-tumor effect of anthracyclines is accompanied by dose-dependent cardiac toxicity that has been thoroughly investigated in studies trying to establish a safe dose of anthracyclines ( Cardiotoxicity of DOX clinically presents as both acute and chronic

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Biomimetic nanoflowers by self-assembly of nanozymes to induce intracellular oxidative damage against hypoxic tumors

the reduction in O 2 to H 2 O 2 is carried out in four steps during which occur ROS and are as follows: 1

Higher concentrations (3%+) are best reserved for experienced users or targeted use, as they raise the risk of irritation and staining without clearly proportional benefits