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The practical focus is glucose control, IGF-1, liver/kidney context, lipids, and cardiovascular symptoms

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5, SeNPs exert their anti-ferroptotic effects in renal tubular epithelial cells through a stepwise mechanism: they are first internalized into the cytoplasm via endocytosis, subsequently enriched within lysosomes, and then stimulate XBP1 expression to block I/R-induced excessive activation of NCOA4-mediated ferritinophagy, ultimately interrupting iron-dependent cell death cascades

We have also tried to address this in our rewrite of the therapeutics section found in the second half of the paper.