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It has been demonstrated that addressing the deficit in synaptic plasticity caused by neuronal atrophy and cell death can be beneficial in the treatment of depression ( Currently, the primary focus of clinical depression treatment lies in targeting monoamine neurotransmitters

Delivery of insulin-like growth factor-I to the rat brain and spinal cord along olfactory and trigeminal pathways following intranasal administration

Physical daily activity increases blood circulation and neurotransmitters

A full description of the mathematical model is given