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Conclusion TMQ0153 demonstrates significant potential as a therapeutic strategy for AML, particularly by targeting mitochondrial function and overcoming treatment resistance

As the owner of two MTHFR gene variations, I can surely attest to this

I think we can edit this part while I'm spacing out

And then of course, there's going to be the, I don't want to call it placebo, but there's going to be the context, the conditioned place context of it

Additionally, it also prevented depolarization of the mitochondrial membrane and suppressed mitochondrial swelling and production of mitochondrial ROS