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The FDA label recommends monitoring for glucose intolerance and IGF-1 levels

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Though the precise mechanism of action is not fully elucidated, evidence from coarse-grained molecular dynamics and high-content imaging suggests a concentration-dependent, multimodal transcellular mechanism, partly mediated by its surfactant-like properties (Hossain et al., 2020

They suppress the FoxO3a-MuRF1/Atrogin-1 axis and TNF-/NF-B inflammatory pathways, ultimately preserving mitochondrial function and muscle integrity (Bellanti et al., 2022)

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