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After adding either the cGASSTING pathway inhibitor C-176 or GLP-1RAs, the cGAS-STING signaling pathway was inhibited effectively accompanied with suppression of chondrocyte ferroptosis, inflammatory factor expression, and osteophyte formation on articular surfaces, and enhancement of cartilage repair, reinforcing a causal link between cGASSTING activation and chondrocyte ferroptosis in RA

Rather than viewing complexity as an inherent liability, contemporary pharmacology increasingly recognizes that pleiotropic signaling can be therapeutically advantageous when appropriately modulated through dosing, bias, and patient stratification (Kenakin, 2019

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