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Lane 1 is endogenous stimulation via diet and nutrients

It is diagnosed in around 60% of these patients (119)

In contrast to glutamate oxidation via GDH, that by the aspartate transaminase also requires use of oxaloacetate, in the proposed model generated during glutamate production (labeled 4 close to the lower edge of Figure 3B)

Patients who have developed these conditions often report that the symptoms began after they started taking a GLP-1 medication and, in many cases, persisted even after they stopped using the drug

With the breakthroughs in metabolomics technology, the research on epilepsy and metabolism has entered a systematic stage, and their relationship has attracted increasing attention

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