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These peptide segments are usually designed to have specific biological activities (e.g., the GLP-1 receptor agonist in MariTide) for interaction with target molecules

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Given that KRAS mutations are prevalent in almost 30% of non-small cell lung cancer (NSCLC) patients [2], we chose a Kras G12D -driven spontaneous lung cancer murine model to elucidate the detailed mechanism by which DDX3X regulates lung cancer progression, which might be also extended to KRAS wildtype lung cancers

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The amount of muscle loss varies by individual and correlates with total weight reduction

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