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GHK-Cu is understood to upregulate collagen synthesis in dermal fibroblasts, a mechanism explored in multiple studies, including research on copper peptides and skin regeneration published in Biomolecules , which also examined GHK's broader gene-modulating effects

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By repressing cardiac peroxisome proliferator-activated receptor-, miR-199a facilitates a metabolic shift from a predominant reliance on fatty acid utilization toward an increased reliance on glucose metabolism, which leads to lethal heart failure (36)

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