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Glutathione reductase (GR) reduces glutathione disulfide (GSSG) and drives the GSH-dependent systems [7,8], whereas Trx reductases (TrxR) are pivotal in the Trx-dependent system (Figure 1) [9]
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Without adequate glutathione to support detoxification, these toxic Phase I intermediates would accumulate, causing massive oxidative stress, mitochondrial dysfunction, and ultimately, severe liver damage
Because PTSD pathophysiology is so closely related to ROS homeostasis, it is important to analyze the processes related to ROS production, specifically within the mitochondria (Lushchak et al., 2023)
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However, fenofibrate may affect ovarian estrogen synthesis and induce hepatomegaly in mice, and its long-term safety and hormonal effects are of concern [359]
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