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While direct mutagenic effects were not identified in safety studies, proliferative effects were, representing a plausible pathway for increased risk of PDAC

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730 FtMt protein expression is significantly upregulated in MCAO/R mice, and the absence of FtMt promotes free iron deposition and lipid peroxidation, which aggravates ferroptosis and brain injury caused by MCAO/R

doi: 10.1024/0300-9831.79.3.131.

K., Cristofaro, V., & Sullivan, M